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tional and emotional speech (see above) The patient recognizes his ineptitude and mistakes Repeated failures in speech cause exasperation or despair As a result of damage to the adjacent prerolandic motor area, the arm and lower part of the face are usually weak on the right side, and occasionally the leg as well The tongue may deviate away from the lesion, ie, to the right, and be slow and awkward in rapid movements For a time, despite the relative preservation of auditory comprehension and the ability to read, commands to purse, smack, or lick the lips or to blow and whistle and make other purposeful movements are poorly executed, which signi es that an apraxia has extended to certain other learned acts involving the lips, tongue, and pharynx In these circumstances, imitation of the examiner s actions with orofacial muscles is performed better than execution of acts on command Self-initiated actions, by contrast, may be normal By implication from imaging ndings in patients with apractic speech, the co-ordination of orobuccolingual movements, which are responsible for articulation, takes place in the left insular cortex (see Dronkers) rather than primarily in the Broca s area Scanning by positron emission tomography (PET) shows activation of the insular region as well as of the lateral premotor cortex and the anterior pallidum during repetition of single words (Wise et al) It would be incorrect, however, to conclude from these studies that Broca s area itself does not participate in the composition of articulatory movements during natural speech

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In the milder form of Broca s aphasia and in the recovery phase of the severe form, patients are able to speak aloud to some degree, but the normal melody of speech is entirely lacking Words are uttered slowly and laboriously and enunciated poorly Missing is the normal in ection, intonation, phrasing of words in a series, and pacing of word utterances The overall impression is one of a lack of uency, a term that has come to be almost synonymous with aphasias that derive from damage in and around the Broca area This labored, unin ected speech stands in contrast to the uent speech of Wernicke s aphasia Speech in Broca s aphasia is sparse (10 to 15 words per minute as compared with the normal 100 to 115 words per minute) and consists mainly of nouns, transitive verbs, or important adjectives; phrase length is abbreviated and many of the small words (articles, prepositions, conjunctions) are omitted, giving the speech a telegraphic character (so-called agrammatism) The substantive content of the patient s language permits the crude communication of ideas, sometimes despite gross expressive dif culties Repetition of the examiner s spoken language is as abnormal as the patient s own speech If a patient with non uent Broca s aphasia has no dif culty in repetition, the condition is termed transcortical motor aphasia (see further on) Most patients with Broca s aphasia have a correspondingly severe impairment in writing Should the right hand be paralyzed, the patient cannot print with the left one, and if the right hand is

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spared, the patient fails as miserably in writing to request and replying to questions in written form Letters are malformed and words misspelled While writing to dictation is impossible, letters and words can still be copied The dysgraphia usually corresponds in degree to the severity of the spoken disturbance, but there are exceptions in which one is far more affected The comprehension of spoken and written language, though seemingly normal under casual conditions, is usually defective in Broca s aphasia and will break down under stringent testing, especially when novel or complicated material is introduced The naming of objects and particularly parts of objects is particularly faulty These are the most variable and controversial aspects of Broca s aphasia, since in some patients with a loss of motor speech and agraphia as a result of cerebral infarction, the understanding of spoken and written language may be virtually normal Mohr has pointed out that in such patients an initial mutism is usually replaced by a rapidly improving dyspraxic and effortful articulation, leading to complete recovery (a mini-Broca s aphasia, in his terms) The lesion in such cases is restricted to a zone in and immediately around the posterior part of the inferior frontal convolution (Broca s area per se) Mohr has stressed the distinction between this relatively mild and restricted type of motor speech disorder and the more complex syndrome that is traditionally referred to as Broca s aphasia The lesion in the major form of Broca s aphasia is much larger than originally described, involving not only the inferior frontal gyrus but also the subjacent white matter and even the head of the caudate nucleus and putamen (Fig 23-2), the anterior insula, frontoparietal operculum, and adjacent

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